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  Indian J Med Microbiol
 

Figure 2: Changes in neural activity in response to highly salty stimuli. (a) Grand averaged event-related potentials over all subjects in response to 200 mM NaCl on channel Cz (n = 7), and scalp distributions at the 2 time points indicated by the shaded areas including N1 (120–180 ms after stimulus onset) and P2 (160–250 ms after stimulus onset) components. Please note that different scales are used. (b) Scatter plot showing the correlation between the peak amplitudes of the N1 component from the respective electrodes and the individual's salt intake/preference under either the 150 mM NaCl stimulus (left, n = 6) or the 200 mM NaCl stimulus (right, n = 7). The solid lines in the scatter plot indicate the regression line for each of the respective electrodes (line of best fit). (c) Dipole modeling of the intracranial sources of the N1 component (over the interval 120–180 ms) with the 150 mM NaCl stimulus (left two images). PCA indicates that two principal components accounted for 87.3% of the variance. The first dipole is located in the OFC (Talairach coordinates: x, y, z = −46.4, 39, −6.3) and the second covered the insula area (Talairach coordinates: x, y, z = −56.5, −3.6, 4.8). Source analysis was also performed on the EEG data of the N1 component across subjects using SPM (right two images). Group inversion was used in the three dimensional source reconstruction, and the rendered view of the grand mean file is presented here

Figure 2: Changes in neural activity in response to highly salty stimuli. (a) Grand averaged event-related potentials over all subjects in response to 200 mM NaCl on channel Cz (<i>n</i> = 7), and scalp distributions at the 2 time points indicated by the shaded areas including N1 (120–180 ms after stimulus onset) and P2 (160–250 ms after stimulus onset) components. Please note that different scales are used. (b) Scatter plot showing the correlation between the peak amplitudes of the N1 component from the respective electrodes and the individual's salt intake/preference under either the 150 mM NaCl stimulus (left, <i>n</i> = 6) or the 200 mM NaCl stimulus (right, <i>n</i> = 7). The solid lines in the scatter plot indicate the regression line for each of the respective electrodes (line of best fit). (c) Dipole modeling of the intracranial sources of the N1 component (over the interval 120–180 ms) with the 150 mM NaCl stimulus (left two images). PCA indicates that two principal components accounted for 87.3% of the variance. The  first dipole is located in the OFC (Talairach coordinates: x, y, z = −46.4, 39, −6.3) and the second covered the insula area (Talairach coordinates: x, y, z = −56.5, −3.6, 4.8). Source analysis was also performed on the EEG data of the N1 component across subjects using SPM (right two images). Group inversion was used in the three dimensional source reconstruction, and the rendered view of the grand mean file is presented here